Introduction. Ramsay Hunt syndrome is a rare neurological condition caused by the reactivation of the varicella-zoster virus (VZV) in the ganglia of the facial nerve. It is characterized by facial paralysis, varioliform eruptions, and severe pain. Objective of the study: to present a clinical case of Ramsay Hunt syndrome in a 7-year-old child. Material and methods. Clinical-anamnestic data, results of clinical and immunological investigations were extracted from the inpatient medical records of the Mother and Child Institute. Results. A 7-year-old child admitted to the neurology department presented with facial asymmetry, left lagophthalmos, left ear pain, and fever of 38°C. The following day, the child was transferred to the emergency ENT department with clinical signs of mastoiditis to undergo left antromastoidotomy. On the third day, confluent vesicular eruptions typical of Herpes Zoster infection appeared in the left periauricular region, which were painful and pruritic. Therapy included systemic antiviral and corticosteroid preparations. Subsequently, Ceftriaxone antibiotic therapy was administered. The child’s condition improved, but on the 7th day, signs of drug eruption appeared. Laboratory tests revealed positive Herpes Zoster IgM, negative Herpes Zoster IgG, and IgE levels of 15 IU/ml. Considering the clinical course with prompt development of unilateral facial paralysis, localized intense periauricular pain, and herpetiform eruptions caused by the Herpes Zoster virus, Ramsay Hunt syndrome was confirmed in the child. Additionally, antihistamine preparations were prescribed. The patient’s condition improved with remission of cutaneous eruptions and improvement of neurological and otolaryngological symptoms. Discharged home after 2 weeks from the onset of the illness. Conclusion. The presented case underscores the importance of understanding and appropriately managing Ramsay Hunt syndrome in children, as although this syndrome predominantly develops in the elderly, isolated cases can occur in the pediatric population. Infection with the Herpes Zoster virus was considered a risk factor for the development of drug allergic reactions during antibiotic therapy.